Overview
Spinal cord injury is damage to the spinal cord that disrupts the transmission of motor, sensory, and autonomic signals between the brain and the body below the level of the lesion. It is classified by anatomical level and by completeness, with complete injuries abolishing function below the lesion and incomplete injuries sparing variable motor or sensory capacity. Injuries arise from traumatic mechanisms, including falls, vehicle collisions, and sport, where equestrian and other high-impact activities carry notable risk, as well as from vascular and structural causes such as arteriovenous fistulae and other lesions affecting cord perfusion. The pathophysiology involves primary mechanical disruption followed by a secondary cascade of ischemia, oxidative stress, inflammation, and cell death that extends tissue damage over time; experimental work on oxidative injury in neural cells and on the molecular biology of oligodendrocytes, myelination, and signaling pathways such as Wnt informs understanding of this progression. Consequences range from paralysis and altered sensation to disturbances of bladder, bowel, and cardiovascular control. Research directions include neuroprotection to limit secondary injury, and regenerative strategies using stem cells, biocompatible scaffolds, and three-dimensional neural culture to support repair and remodeling. Clinically, management combines acute stabilization, prevention of complications, and long-term rehabilitation aimed at maximizing function, independence, and quality of life.
Research published in this journal
9 peer-reviewed articles, ranked by relevance. Each links to its DOI.
Oligodendrocytes Development and Wnt Signaling Pathway
Embolization for Perimedullary Arteriovenous Fistulae: Pioneering Experience in Peru
In Vitro Cytoprotection of Resveratrol against H2O2-Induced Oxidative Stress and Injury in Astrocytes
RBM45: Molecular, Cellular, and Evolutionary Biology
Biocompatible Scaffolds for Human-Induced Pluripotent Stem Cell Transplantation and Modeling Post-Stroke Recovery in Three-Dimensional Neural Cell Culture
Mucosa-Muscular Signaling for Bile-Induced Esophageal Dysmotility. An Experimental Study in Ex-Vivoguinea-Pig Isolated Esophagi
Rbm45 Phylogenetics, Protein Domain Conservation, and Gene Architecture in Clade Metazoa
How this research is being cited
The 9 articles above have been cited 40 times in the scholarly literature. Citation data via OpenAlex and Crossref, updated Jun 2026.
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2026 · International Journal of Innovative Technologies in Social Science
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2026 · Cells
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2026 · Molecular Psychiatry
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2025 · BMC Genomics
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2025 · bioRxiv (Cold Spring Harbor Laboratory)
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2025 · PLOS One
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2025 · Genome Biology
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2025 · PLoS ONE
A sample of recent works citing this journal's research on Spinal Cord Injury, linking to each citing work.