Cerebral Malaria

Overview

Cerebral Malaria is a severe and life-threatening complication of malaria caused by the parasitic infection of the brain. This condition is characterized by the inflammation of the blood vessels in the brain, which can lead to brain damage and even death in some cases. Cerebral Malaria is caused by the Plasmodium falciparum parasite, which is transmitted to humans through the bite of an infected mosquito. The symptoms of this condition include high fever, headache, confusion, seizures, and loss of consciousness. The early symptoms of cerebral malaria may be similar to those of other types of malaria, but they can rapidly progress to life-threatening complications. The treatment of cerebral malaria includes prompt diagnosis and administration of antimalarial drugs. In severe cases, hospitalization may be required for supportive care like oxygen, fluids and electrolytes. Furthermore, prompt and appropriate management of other clinical manifestations such as seizures, high blood sugar, or low sodium and potassium levels may be helpful in preventing complications. Preventive measures are also important in the prevention of cerebral malaria. These include using insecticide-treated bed nets, wearing protective clothing, and the use of malaria prophylaxis medications while traveling to malaria-endemic areas. In conclusion, Cerebral Malaria is a serious complication of malaria that can lead to severe brain damage and even death. Prompt diagnosis and treatment can help prevent complications and improve outcomes in patients with this condition, and preventive measures are important in preventing the spread of this disease.

Research published in this journal

1 peer-reviewed article, ranked by relevance. Each links to its DOI.

How this research is being cited

The 1 article above has been cited 7 times in the scholarly literature. Citation data via OpenAlex and Crossref, updated Jun 2026.

• Suppression of 8-oxoguanine DNA glycosylase (OGG1) activity produced positive impacts on disease severity, survival, and histopathological features of mice infected with Plasmodium berghei.
  Mukhtar Gambo Lawal et al. · 2025 · Experimental parasitology
• Suppression of 8-oxoguanine DNA glycosylase (OGG1) activity produced positive impacts on disease severity, survival, and histopathological features of mice infected with Plasmodium berghei
  2025 · Experimental Parasitology
• Vitamin C-rich juice co-administration with artemether-lumefantrine ameliorates oxido-inflammatory responses in Plasmodium berghei-infected mice
  A. Ajayi et al. · 2023 · Parasitology Research
• Vitamin C-rich juice co-administration with artemether-lumefantrine ameliorates oxido-inflammatory responses in Plasmodium berghei-infected mice
  2023 · Parasitology Research
• Cord Malaria Infection, Complement Activation, Oxidative Stress, Gestational Age, and Birth Weight, Characterized by High Plasmodium falciparum Prevalence in Bamenda, Cameroon
  Oumar Mahamat et al. · 2020 · Journal of Tropical Medicine
• Cord Malaria Infection, Complement Activation, Oxidative Stress, Gestational Age, and Birth Weight, Characterized by High Plasmodium falciparum Prevalence in Bamenda, Cameroon
  2020 · Journal of Tropical Medicine
• Regulation of Expression of Reactive Oxygen Intermediates During Plasmodium Infection to Reduce Immunopathology Provides a Possible Antioxidant Adjuvant to Enhance Anti-Malarial Drug Therapy
  2017 · International Journal of Clinical Microbiology

A sample of recent works citing this journal's research on Cerebral Malaria, linking to each citing work.

This page summarises published research for orientation; it is not medical or professional advice.

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