High incidence of amyotrophic lateral sclerosis (ALS) occurred among the Chamorro natives in Guam back in the 1940s and 1950s, leading scientists to link cyanobacteria and one of its neurotoxin, beta-N-methyl amino-L-alanine (BMAA) to ALS. Subsequent findings of the regular biosynthesis of BMAA in the Baltic Sea combined with its possible transfer and bioaccumulation within major food webs, some ending in human consumption, has been alarming 1. A prevailing theory has been that long term, chronic exposure to low levels of BMAA through the environment, in areas with algae blooms, occurs through biomagnification which might cause ALS in genetically predisposed individuals. Recent studies from Scandinavia have been far less supportive, finding limited evidence for the theory among retail coastal seafood. BMAA was identified in blue mussel, oyster, shrimp, plaice, char and herring, but was undetectable in other samples (salmon, cod, perch and crayfish) casting doubt on biomagnification in many seafood networks humans eat 2. Others are more skeptical of any cyanobacteria or BMAA connection and believe poor reporting and analysis and prolific errors have weakened the research 3. Yet, research from plausible, rational studies showing a possible link have been published in credible journals.