University of Catania, Department of Clinical and Molecular Biomedicine, Catania
University of Catania, Department of Clinical and Molecular Biomedicine, Catania.
Medicine and Life Scinces.
- microRNAs in Parkinson’s Disease: From Pathogenesis to Novel Diagnostic and Therapeutic Approaches.
- Wnt3a promotes pro-angiogenic features in macrophages in vitro : Implications for stroke pathology.
- Nigrostriatal degeneration dictates a top-down genetic program for neuroplasticity and neurorepair: Focus on the hippocampus and Wnt/GSK-3β/β-catenin signaling cascade.
- Wnt/β-Catenin Signaling Is Required to Rescue Midbrain Dopaminergic Progenitors and Promote Neurorepair in Ageing Mouse Model of Parkinson's Disease.
- The role of the immune system in central nervous system plasticity after acute injury.
- Targeting Wnt signaling at the neuroimmune interface for dopaminergic neuroprotection/repair in Parkinson's disease.
- Uncovering novel actors in astrocyte-neuron crosstalk in Parkinson's disease: The Wnt/β-catenin signaling cascade as the common final pathway for neuroprotection and self-repair.
- Reactive Astrocytes Are Key Players in Nigrostriatal Dopaminergic Neurorepair in the Mptp Mouse Model of Parkinson’s Disease: Focus on Endogenous Neurorestoration.
- Aging-Induced Nrf2-ARE Pathway Disruption in the Subventricular Zone Drives Neurogenic Impairment in Parkinsonian Mice via PI3K-Wnt/ -Catenin Dysregulation.
- Plasticity of Subventricular Zone Neuroprogenitors in MPTP (1-Methyl-4-Phenyl-1,2,3,6-Tetrahydropyridine) Mouse Model of Parkinson's Disease Involves Cross Talk between Inflammatory and Wnt/ -Catenin Signaling Pathways: Functional Consequences for Neuroprotection and Repair.
- Switching the Microglial Harmful Phenotype Promotes Lifelong Restoration of Subtantia Nigra Dopaminergic Neurons from Inflammatory Neurodegeneration in Aged Mice.
- A Wnt1 Regulated Frizzled-1/beta-Catenin Signaling Pathway as a Candidate Regulatory Circuit Controlling Mesencephalic Dopaminergic Neuron-Astrocyte Crosstalk : Therapeutical Relevance for Neuron Survival and Neuroprotection.