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United States

Yale University | YU · Yale Cancer Center-Section of Hematology.

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Oscar Ramirez


Yale University | YU
Yale Cancer Center-Section of Hematology
New Haven, CT.

Research Interests:

  • PCR
  • Cell Culture
  • Gel Electrophoresis
  • Gene Expression
  • Western Blot Analysis
  • Cloning
  • Cancer Biology
  • Flow Cytometry
  • Immunohistochemistry
  • Cell Signaling
  • Molecular Cloning
  • Apoptosis.


  • Oscar Ramirez currently works at the Yale Cancer Center-Section of Hematology, Yale University. 
  • Oscar does research in Immunology, Cell Biology and Molecular Biology. 
  • Their most recent publication is 'E2F-2 Promotes Nuclear Condensation and Enucleation of Terminally Differentiated Erythroblasts..'


  • E2F-2 Promotes Nuclear Condensation and Enucleation of Terminally Differentiated Erythroblasts.
  • Small interfering RNA targeting NF-κB attenuates lipopolysaccharide-induced acute lung injury in rats.
  • Cancer-associated SF3B1 mutants recognize otherwise inaccessible cryptic 3' splice sites within RNA secondary structures.
  • Time Series Analyses of Hand, Foot and Mouth Disease Integrating Weather Variables
  • Correction: A Small Library of Synthetic Di-Substituted 1, 4-Naphthoquinones Induces ROS-Mediated Cell Death in Murine Fibroblasts
  • Leptin deficiency in vivo enhances the ability of splenic dendritic cells to activate T cells
  • Myeloid-derived suppressor cells are associated with disease progression and decreased overall survival in advanced-stage melanoma patients
  • Abstract A98: Young women's breast cancer demonstrates increased immune suppression through ciculating regulatory T cells and myeloid-derived supressor cells independent of stage or subtype.
  • Abstract P4-04-06: Young women's breast cancer is characterized by increased immune suppression through circulating myeloid derived supressor cells
  • Influence of human immune cells on cancer: Studies at the University of Colorado
  • Abstract 5405: Immuno-phenotyping reveals cell types present in circulation of breast cancer patients and elucidates their funstional potential
  • Implication for the role of leptin-induced signaling as a negative regulator of dendritic cell function.